简介:摘要:变电站的自动技术最近的历史到20多年,随着智能化电子设备的逐渐普及,单向的自动化系统大量形成。自动化系统单元之间各自独立,并没有建立直接的电气联系,又由于变电站的电力系统隶属于企业内的不同部门,其管理和维护的种类繁多导致性能,可靠性和系统运行成本都成倍增加,自动化孤岛问题困扰着电力系统的改革进程。国际电工委员会提出了IEC61850规约作为统一的标准和规范,从而能基于网络通信体系的基础,电力系统设备涉及到通信网络、接口映射等多方面内容,能够提供高层次的信息共享机制,实现可靠快速通信保障。
简介:摘要:本文以600MW火电机组的开关设备和电缆选择工程实例为基础,介绍了IEC60909标准短路计算的等效电网、变压器、发电机、异步电动机等模型和计算方法,同时也介绍了一个严格遵循IEC60909标准的短路计算软件ETAP。
简介:AbstractPurpose:The reliability of trauma coding is essential in establishing the reliable trauma data and adopting efficient control and monitoring policies. The present study aimed to determine the reliability of trauma coding in educational hospitals affiliated to Shahid Beheshti University of Medical Sciences, Iran.Methods:In this descriptive cross-sectional study, 591 coded medical records with a trauma diagnosis in 2018 were selected and recoded by two coders. The reliability of trauma coding was calculated using Cohen's kappa. The data were recorded in a checklist, in which the validity of the content had been confirmed by experts.Results:The reliability of the coding related to the nature of trauma in research units was 0.75—0.77, indicating moderate reliability. Also, the reliability of the coding of external causes of trauma was 0.57—0.58, suggesting poor reliability.Conclusion:The reliability of trauma coding both in terms of the nature of trauma and the external causes of trauma does not have a good status in the research units. This can be due to the complex coding of trauma, poor documentation of the cases, and not studying the entire case. Therefore, holding training courses for coders, offering training on the accurate documentation to other service providers, and periodically auditing the medical coding are recommended.
简介:【摘要】本文作者结合自己多年的工作经验对继电保护的现状进行分析,并对系统中应配置的继电 保护装置及其配置情况进行表述 ,提出了应采用不同的逻辑保护应用到不同负荷的变配电系统。
简介:摘要目的探索60Co γ射线诱导大鼠小肠上皮细胞(IEC-6)中CPT1A和CPT1B蛋白表达变化,并进一步研究肉碱棕榈酰转移酶1(CPT1)变化对受照细胞增殖的影响及相关分子机制。方法IEC-6细胞经棕榈酸、血清饥饿以及血清饥饿联合棕榈酸处理后,给予0、5、10或15 Gy 60Co γ射线照射,照射后24 h收集细胞并提取蛋白,利用Western blot方法检测CPT1A和CPT1B蛋白的表达水平变化;ETO是CPT1的小分子抑制剂,利用克隆形成率实验和CCK-8实验分析ETO抑制CPT1对60Co γ射线照射IEC-6细胞存活及增殖的影响;利用Western blot方法检测5 Gy 60Co γ射线照射ETO处理IEC-6细胞48 h后细胞外信号调节激酶1/2(ERK1/2)、c-Jun氨基末端激酶(JNK)蛋白表达及磷酸化水平变化。结果棕榈酸处理组中,CPT1A蛋白在15 Gy γ射线照射后表达量显著增加(t=-2.82,P<0.05)。血清饥饿处理组中,CPT1A蛋白在5、10和15 Gy γ射线照射后表达量明显升高(t=-3.28、-8.72、-8.67,P<0.05);血清饥饿联合棕榈酸处理组中,CPT1A蛋白在5、10和15 Gy γ射线照射后表达量显著增加(t=-10.69、-7.02、-8.23, P<0.05),CPT1B蛋白在照射10和15 Gy γ射线照射后表达量显著增加(t=-3.73、-5.05, P<0.05)。60Co γ射线照射后,ETO处理组细胞存活率及相对增殖率明显低于对照组(t=5.46、13.22,P<0.05);ERK1/2蛋白表达水平及JNK磷酸化水平明显低于对照组(t=4.01、3.29、10.68、14.44,P<0.05)。结论CPT1通过促进增殖通路中ERK1/2蛋白的表达和JNK的激活,从而使得60Co γ射线诱导IEC-6细胞损伤后的存活及增殖增加。