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简介：我们检验了在巴西半干旱的区域的三大西洋福雷斯特残余与Vismiaguianensis叶垃圾联系的mycobiota。在学习地点之中，二残余是保护的森林储备，而第三被主要人为的活动影响。十八件崽样品被收集在湿并且旱季并且被粒子过滤处理技术。一个总数4750真菌142孤立taxa被识别。种类丰富在在湿季节期间收集的崽样品是更高的。Nonmetric多维的可伸缩multivariate分析在采样地点和季节之中在真菌的社区的作文显示出差别。类似的分析证明差别是统计上重要的(R?=?0.85；P?=?0.0001)。我们的调查结果揭示了那空间、时间的异质，并且人的活动在V的saprobic真菌上有重要影响。guianensis叶垃圾。

简介：Close-rangehyperspectralimagesareapromisingsourceofinformationinplantbiology,inparticular,forinvivostudyofphysiologicalchanges.Inthisstudy,weinvestigatehowdatafusioncanimprovethedetectionofleafelementsbycombiningpixelreflectanceandmorphologicalinformation.Thedetectionofimageregionsassociatedtotheleafstructuresisthefirststeptowardquantitativeanalysisonthephysicaleffectsthatgeneticmanipulation,diseaseinfections,andenvironmentalconditionshaveinplants.WetestedourfusionapproachonMusaacuminata(banana)leafimagesandcompareditsdiscriminantcapabilitytosimilartechniquesusedinremotesensing.Experimentalresultsdemonstratetheefficiencyofourfusionapproach,withsignificantimprovementsoversomeconventionalmethods.

简介：Pomegranateleaf(PGL)hasadefiniteroleinregulatinglipidmetabolism.However,pharmacokineticresultsshowthemainactiveingredient,ellagicacid,inPGLhasloweroralbioavailability,suggestingthatthelipid-loweringeffectofPGLmayactthroughinhibitinglipidabsorptioninthesmallintestine.Ourresultsdemonstratedthatpomegranateleafanditsmainactiveingredients(i.e.,ellagicacid,gallicacid,pyrogallicacidandtannicacid)werecapableofinhibitingpancreaticlipaseactivityinvitro.Incomputationalmoleculardocking,thefouringredientshadgoodaffinityforpancreaticlipase.AcutelipidoverloadexperimentsshowedthatalargedosageofPGLsignificantlyreducedserumtotalcholesterol(TG)andtriglycerides(TC)levelsinadditiontoinhibitingintestinallipaseactivity,whichdemonstratedthatPGLcouldinhibitlipaseactivityandreducetheabsorptionoflipids.WealsofoundthatPGLcouldreversethereducedtight-junctionproteinexpressionduetointestinallipidoverload,promoteOccludinandClaudin4expressioninthesmallintestine,andenhancetheintestinalmucosalbarrier.Inconclusion,wedemonstratedthatPGLcaninhibitlipidabsorptionandreducebloodTGandTCbytargetingpancreaticlipase,promotingtight-junctionproteinexpressionandtherebypreventingintestinalmucosadamagefromanoverloadoflipidsintheintestine.